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The Peri‐Implantitis: Implant Surfaces, Microstructure, and Physicochemical Aspects

Identifieur interne : 000074 ( France/Analysis ); précédent : 000073; suivant : 000075

The Peri‐Implantitis: Implant Surfaces, Microstructure, and Physicochemical Aspects

Auteurs : Jaafar Mouhyi [Suède] ; David M. Dohan Ehrenfest [France] ; Tomas Albrektsson [Suède]

Source :

RBID : ISTEX:DD9687F71B65EDDBF6C2DD900C0DB243DBA1C962

Descripteurs français

English descriptors

Abstract

There are two ways of looking at secondary failures of osseointegration; one is to reflect on possible causes for the failure, the other focuses on the pathology per se. In the first case, background factors such as mechanical trauma (adverse loading) or inflammations/infections are being discussed as the cause of failure. Then peri‐implantitis is a term reserved for implant disturbance due to inflammation/infections only. However, irrespective of the original reason for the failure being adverse loading or inflammation/infection, the end result with bone resorption and inflammation may be very similar. Hence, in the present article, an alternative outlook has been chosen. Trigerring factors for peri‐implantitis are generally gathered under four categories: lesions of peri‐implant attachment, presence of aggressive bacteria, excessive mechanical stress, and corrosion. If only one of these factors would start a chain reaction leading to lesions, then the other factors may combine to worsen the condition. With other words, peri‐implantitis is a general term dependent on a synergy of several factors, irrespective of the precise reason for first triggering off symptoms.

Url:
DOI: 10.1111/j.1708-8208.2009.00244.x


Affiliations:


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ISTEX:DD9687F71B65EDDBF6C2DD900C0DB243DBA1C962

Le document en format XML

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<term>Antibiotic</term>
<term>Antimicrobial therapy</term>
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<term>Beagle dogs</term>
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<term>Dohan ehrenfest</term>
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<term>Physicochemical mechanisms</term>
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<term>Prosthet dent</term>
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<term>Sahlgrenska academy</term>
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<term>Soft tissue</term>
<term>Superoxide radicals</term>
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<term>Temperature increase</term>
<term>Titanium</term>
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<term>Absolute ethanol</term>
<term>Abutment screw</term>
<term>Adverse loading</term>
<term>Antibiotic</term>
<term>Antimicrobial therapy</term>
<term>Atomic composition</term>
<term>Beagle dogs</term>
<term>Berglundh</term>
<term>Biocompatibility</term>
<term>Biomaterial</term>
<term>Biomaterial applications</term>
<term>Biomaterials</term>
<term>Biomed</term>
<term>Biomed mater</term>
<term>Bone grafts</term>
<term>Bone regeneration</term>
<term>Bone resorption</term>
<term>Bone response</term>
<term>Bone tissues</term>
<term>Brous tissue</term>
<term>Carbon dioxide laser</term>
<term>Chain reaction</term>
<term>Citric</term>
<term>Citric acid</term>
<term>Citric acid treatment</term>
<term>Clean surface</term>
<term>Clin</term>
<term>Clin implant dent relat</term>
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<term>Different oxide thicknesses</term>
<term>Dohan ehrenfest</term>
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<term>Experimental study</term>
<term>Foreign body</term>
<term>Free radicals</term>
<term>Global treatment</term>
<term>Granulation tissue</term>
<term>Growth factors</term>
<term>Hydrogen peroxide</term>
<term>Implant</term>
<term>Implant dent</term>
<term>Implant failures</term>
<term>Implant surface</term>
<term>Implant surfaces</term>
<term>Interface</term>
<term>Laser</term>
<term>Laser beam</term>
<term>Lausmaa</term>
<term>Lesion</term>
<term>Light microscopy</term>
<term>Lindhe</term>
<term>Lob5 foundation</term>
<term>Mater</term>
<term>Maxillofac</term>
<term>Mouhyi</term>
<term>Nonmobile apical part</term>
<term>Oral maxillofac implants</term>
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<term>Other hand</term>
<term>Oxide</term>
<term>Oxide growth</term>
<term>Oxide structure</term>
<term>Oxide thickness</term>
<term>Oxygen source</term>
<term>Oxygenated derivatives</term>
<term>Pathological equilibrium</term>
<term>Periimplantitis</term>
<term>Periimplantitis lesions</term>
<term>Periodontal</term>
<term>Periodontol</term>
<term>Peroxide</term>
<term>Photoelectron spectroscopy</term>
<term>Physical techniques</term>
<term>Physicochemical</term>
<term>Physicochemical mechanisms</term>
<term>Pilot study</term>
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<term>Prosthet dent</term>
<term>Regeneration</term>
<term>Risk factors</term>
<term>Sahlgrenska academy</term>
<term>Scanning electron microscope</term>
<term>Soft tissue</term>
<term>Superoxide radicals</term>
<term>Surface decontamination</term>
<term>Surface oxide</term>
<term>Temperature increase</term>
<term>Titanium</term>
<term>Titanium implant</term>
<term>Titanium implants</term>
<term>Titanium oxide</term>
<term>Titanium oxide layer</term>
<term>Titanium surfaces</term>
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<term>Weakest part</term>
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<front>
<div type="abstract" xml:lang="en">There are two ways of looking at secondary failures of osseointegration; one is to reflect on possible causes for the failure, the other focuses on the pathology per se. In the first case, background factors such as mechanical trauma (adverse loading) or inflammations/infections are being discussed as the cause of failure. Then peri‐implantitis is a term reserved for implant disturbance due to inflammation/infections only. However, irrespective of the original reason for the failure being adverse loading or inflammation/infection, the end result with bone resorption and inflammation may be very similar. Hence, in the present article, an alternative outlook has been chosen. Trigerring factors for peri‐implantitis are generally gathered under four categories: lesions of peri‐implant attachment, presence of aggressive bacteria, excessive mechanical stress, and corrosion. If only one of these factors would start a chain reaction leading to lesions, then the other factors may combine to worsen the condition. With other words, peri‐implantitis is a general term dependent on a synergy of several factors, irrespective of the precise reason for first triggering off symptoms.</div>
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